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Central Neurogenic Hyperventilation. A 53-year-old male patient presented with dyspnea for the past 1 month. A 69-year-old man complained of breathing too fast for 6 weeks. Based on analysis of this patient and other case reports we propose that central neurogenic hyperventilation is uniquely the result of infiltrative tumors that stimulate pontine respiratory centers and central chemoreceptors. Most cases of tumor-induced CNH result from primary CNS despite their rarity comprising only 4 of primary CNS tumors 23.
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1 Owing to its rarity optimal medical management of CNH in this context is currently unknown. Material and Method The clinical and physiological findings in nine patients who developed severe hyperventilation dur ing the course of acute central nervous system disease are detailed. Conclusion Based on analysis of this patient and other case reports we propose that central neurogenic hyperventilation is uniquely the result of infiltrative tumors that stimulate pontine respiratory centers and central chemoreceptors. Based on analysis of this patient and other case reports we propose that central neurogenic hyperventilation is uniquely the result of infiltrative tumors that stimulate pontine respiratory centers and central chemoreceptors. Introduction ndoscopic third ventriculostomy is a common minimal-invasive neurosurgical procedure performed most frequently for patients with obstructive hydrocephalus secondary to impediment of. We report a 51-year-old female patient who was successfully treated with intravenous fentanyl followed by transdermal fentanyl.
This report describes a patient with CNH due to a brainstem anaplastic astrocytoma who also exhibited disturbances of sleep and ocular motor function.
Conclusion Based on analysis of this patient and other case reports we propose that central neurogenic hyperventilation is uniquely the result of infiltrative tumors that stimulate pontine respiratory centers and central chemoreceptors. Central neurogenic hyperventilation CNH for which there is no effective therapy can eventually result in respiratory fatigue and death. Tumor-induced central neurogenic hyperventilation CNH is exceedingly rare. CNH is a syndrome comprising normal or elevated arterial oxygen tension decreased arterial carbon dioxide tension and respiratory alkalosis in the absence of card. Since its first description by Plum and Swanson in comatose patients central neurogenic hyperventilation CNH is increasingly reported in conscious patients with CNS neoplasm. CNH is defined by hyperventilation with low arterial PaCO2 high arterial PaO2 and high arterial pH without drug or metabolic causes 12.
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Central neurogenic hyperventilation CNH for which there is no effective therapy can eventually result in respiratory fatigue and death. This report describes a patient with CNH due to a brainstem anaplastic astrocytoma who also exhibited disturbances of sleep and ocular motor function. CNH is a syndrome comprising normal or elevated arterial oxygen tension decreased arterial carbon dioxide tension and respiratory alkalosis in the absence of card. Introduction ndoscopic third ventriculostomy is a common minimal-invasive neurosurgical procedure performed most frequently for patients with obstructive hydrocephalus secondary to impediment of. Objective To describe a patient with central neurogenic hyperventilation caused by an infiltrative brainstem lymphoma.
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A 69-year-old man complained of breathing too fast for 6 weeks. His consciousness was clear and had bilateral upward gazing palsy. Central neurogenic hyperventilation saline irrigation. Objective To describe a patient with central neurogenic hyperventilation caused by an infiltrative brainstem lymphoma. Central neurogenic hyperventilation often associated with brainstem lymphoma rare in noncomatose patients and never described in CLIPPERS results from released respiratory inhibition from medial pontine nuclear damage.
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Central neurogenic hyperventilation CNH is a rare and easily forgotten diagnosis. Objective To describe a patient with central neurogenic hyperventilation caused by an infiltrative brainstem lymphoma. Abnormal patterns of rate and rhythm are also often a reflection of impaired automatic ventilatory control8 Primary central neurogenic hyperventilationis a rare condition characterised by rapid regular hyperventilation which persists in the face of alkalosis raised oxygen tension low carbon dioxide tension and in the absence of any pulmonary or airway disorder9 10. Central neurogenic hyperventilation CNH for which there is no effective therapy can eventually result in respiratory fatigue and death. Most cases of tumor-induced CNH result from primary CNS despite their rarity comprising only 4 of primary CNS tumors 23.
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Being comfortably tachypneic can be due to a. It is usually due to the midbrain and upper pons damage. She had a transient. 1 Owing to its rarity optimal medical management of CNH in this context is currently unknown. CNH is a syndrome comprising normal or elevated arterial oxygen tension decreased arterial carbon dioxide tension and respiratory alkalosis in the absence of card.
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Central neurogenic hyperventilation CNH is a rare clinical condition that is sometimes difficult to treat. It is usually due to the midbrain and upper pons damage. His consciousness was clear and had bilateral upward gazing palsy. Abnormal patterns of rate and rhythm are also often a reflection of impaired automatic ventilatory control8 Primary central neurogenic hyperventilationis a rare condition characterised by rapid regular hyperventilation which persists in the face of alkalosis raised oxygen tension low carbon dioxide tension and in the absence of any pulmonary or airway disorder9 10. A 53-year-old male patient presented with dyspnea for the past 1 month.
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CNH is a syndrome comprising normal or elevated arterial oxygen tension decreased arterial carbon dioxide tension and respiratory alkalosis in the absence of card. We report a case of a bright alert patient with central neurogenic hyperventilation CNH associated with cerebral malignant lymphoma. Being comfortably tachypneic can be due to a. Most cases of tumor-induced CNH result from primary CNS despite their rarity comprising only 4 of primary CNS tumors 23. Furthermore the hyper-ventilation appears to be directlyneurogenic resulting from damage to specific regions in the central brain stem.
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Six weeks prior to presentation the patient noted an. Material and Method The clinical and physiological findings in nine patients who developed severe hyperventilation dur ing the course of acute central nervous system disease are detailed. Central neurogenic hyperventilation is persistent hyperventilation typically caused by head trauma severe brain hypoxia or lack of cerebral perfusion. Six weeks prior to presentation the patient noted an. Introduction ndoscopic third ventriculostomy is a common minimal-invasive neurosurgical procedure performed most frequently for patients with obstructive hydrocephalus secondary to impediment of.
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We report a 51-year-old female patient who was successfully treated with intravenous fentanyl followed by transdermal fentanyl. The CNH responded clinically to morphine sulfate and. This report describes a patient with CNH due to a brainstem anaplastic astrocytoma who also exhibited disturbances of sleep and ocular motor function. It is usually due to the midbrain and upper pons damage. Central neurogenic hyperventilation CNH is a rare and easily forgotten diagnosis.
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Central neurogenic hyperventilation is persistent hyperventilation typically caused by head trauma severe brain hypoxia or lack of cerebral perfusion. CNH is a syndrome comprising normal or elevated arterial oxygen tension decreased arterial carbon dioxide tension and respiratory alkalosis in the absence of card. Central neurogenic hyperventilation is persistent hyperventilation typically caused by head trauma severe brain hypoxia or lack of cerebral perfusion. Most cases of tumor-induced CNH result from primary CNS despite their rarity comprising only 4 of primary CNS tumors 23. Being comfortably tachypneic can be due to a.
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Central neurogenic hyperventilation is persistent hyperventilation typically caused by head trauma severe brain hypoxia or lack of cerebral perfusion. We report a 51-year-old female patient who was successfully treated with intravenous fentanyl followed by transdermal fentanyl. This report describes a patient with CNH due to a brainstem anaplastic astrocytoma who also exhibited disturbances of sleep and ocular motor function. A case report and discussion of pathophysiology. CNH is defined by hyperventilation with low arterial PaCO2 high arterial PaO2 and high arterial pH without drug or metabolic causes 12.
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Here we describe the case of a patient with CNH secondary to brainstem involvement of a high-grade astrocytoma for. This report describes a patient with CNH due to a brainstem anaplastic astrocytoma who also exhibited disturbances of sleep and ocular motor function. Central neurogenic hyperventilation CNH is a rare and easily forgotten diagnosis. Introduction ndoscopic third ventriculostomy is a common minimal-invasive neurosurgical procedure performed most frequently for patients with obstructive hydrocephalus secondary to impediment of. Since its first description by Plum and Swanson in comatose patients central neurogenic hyperventilation CNH is increasingly reported in conscious patients with CNS neoplasm.
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CNH is defined by hyperventilation with low arterial PaCO2 high arterial PaO2 and high arterial pH without drug or metabolic causes 12. CNH is a syndrome comprising normal or elevated arterial oxygen tension decreased arterial carbon dioxide tension and respiratory alkalosis in the absence of card. 1 Owing to its rarity optimal medical management of CNH in this context is currently unknown. Central neurogenic hyperventilation often associated with brainstem lymphoma rare in noncomatose patients and never described in CLIPPERS results from released respiratory inhibition from medial pontine nuclear damage. Central neurogenic hypoventilation occurs when the medulla respiratory centers are not responding to appropriate stimuli.
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19 rows Central neurogenic hyperventilation CNH is defined as hyperventilation caused. Pantelyat A 1 Galetta SL 1 Pruitt A 1. Abnormal patterns of rate and rhythm are also often a reflection of impaired automatic ventilatory control8 Primary central neurogenic hyperventilationis a rare condition characterised by rapid regular hyperventilation which persists in the face of alkalosis raised oxygen tension low carbon dioxide tension and in the absence of any pulmonary or airway disorder9 10. A case report and discussion of pathophysiology. Material and Method The clinical and physiological findings in nine patients who developed severe hyperventilation dur ing the course of acute central nervous system disease are detailed.
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Central neurogenic hyperventilation CNH is a rare and easily forgotten diagnosis. Central neurogenic hyperventilation CNH for which there is no effective therapy can eventually result in respiratory fatigue and death. Central neurogenic hyperventilation CNH for which there is no effective therapy can eventually result in respiratory fatigue and death. Here we describe the case of a patient with CNH secondary to brainstem involvement of a high-grade astrocytoma for. 19 rows Central neurogenic hyperventilation CNH is defined as hyperventilation caused.
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Here we describe the case of a patient with CNH secondary to brainstem involvement of a high-grade astrocytoma for. CNH is defined by hyperventilation with low arterial PaCO2 high arterial PaO2 and high arterial pH without drug or metabolic causes 12. This report describes a patient with CNH due to a brainstem anaplastic astrocytoma who also exhibited disturbances of sleep and ocular motor function. Furthermore the hyper-ventilation appears to be directlyneurogenic resulting from damage to specific regions in the central brain stem. Based on analysis of this patient and other case reports we propose that central neurogenic hyperventilation is uniquely the result of infiltrative tumors that stimulate pontine respiratory centers and central chemoreceptors.
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19 rows Central neurogenic hyperventilation CNH is defined as hyperventilation caused. It is usually due to the midbrain and upper pons damage. She had a transient. 19 rows Central neurogenic hyperventilation CNH is defined as hyperventilation caused. Abnormal patterns of rate and rhythm are also often a reflection of impaired automatic ventilatory control8 Primary central neurogenic hyperventilationis a rare condition characterised by rapid regular hyperventilation which persists in the face of alkalosis raised oxygen tension low carbon dioxide tension and in the absence of any pulmonary or airway disorder9 10.
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Central neurogenic hyperventilation often associated with brainstem lymphoma rare in noncomatose patients and never described in CLIPPERS results from released respiratory inhibition from medial pontine nuclear damage. Being comfortably tachypneic can be due to a. Central neurogenic hyperventilation often associated with brainstem lymphoma rare in noncomatose patients and never described in CLIPPERS results from released respiratory inhibition from medial pontine nuclear damage. Abnormal patterns of rate and rhythm are also often a reflection of impaired automatic ventilatory control8 Primary central neurogenic hyperventilationis a rare condition characterised by rapid regular hyperventilation which persists in the face of alkalosis raised oxygen tension low carbon dioxide tension and in the absence of any pulmonary or airway disorder9 10. A 53-year-old male patient presented with dyspnea for the past 1 month.
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Central neurogenic hyperventilation is persistent hyperventilation typically caused by head trauma severe brain hypoxia or lack of cerebral perfusion. Central neurogenic hypoventilation occurs when the medulla respiratory centers are not responding to appropriate stimuli. We report a 51-year-old female patient who was successfully treated with intravenous fentanyl followed by transdermal fentanyl. CNH is defined by hyperventilation with low arterial PaCO2 high arterial PaO2 and high arterial pH without drug or metabolic causes 12. Central neurogenic hyperventilation saline irrigation.
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